{"id":1636,"date":"2023-10-11T12:14:31","date_gmt":"2023-10-11T16:14:31","guid":{"rendered":"https:\/\/www.lifetein.com\/blog\/?p=1636"},"modified":"2024-11-16T21:10:45","modified_gmt":"2024-11-17T02:10:45","slug":"alzheimers-disease-and-the-role-of-%ce%b2-amyloid","status":"publish","type":"post","link":"https:\/\/www.lifetein.com\/blog\/alzheimers-disease-and-the-role-of-%ce%b2-amyloid\/","title":{"rendered":"\u03b2-amyloid and Alzheimer\u2019s Disease"},"content":{"rendered":"\n
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The incurable neurodegenerative Alzheimer\u2019s disease has long been associated with \u03b2-amyloid<\/a> build-up in the brain. While this has been known, direct evidence supporting the close relationship of Alzheimer\u2019s disease and the role of \u03b2-amyloid has been hard to come by, until now. Recent A\u03b2-immunotherapy trials have shown that removing aggregated \u03b2-amyloid from symptomatic patients can slow down the disease.

\u03b2-amyloid removal slows the progression of Alzheimer\u2019s disease

This breakthrough holds many implications for future treatment and handling of Alzheimer\u2019s disease. While the new evidence is far from being a cure itself, it presents the opportunity for long-term prevention and potential immunoprevention towards the disease. This is in part due to how early the signs and abnormal \u03b2-amyloid build-up can begin in Alzheimer\u2019s patients, as well as how complex the disease itself can become. However many clinical trials and experiments it may take, groups like LifeTein will always be ready to help supply researchers with the materials they need to make this future possible.

Jucker, M., & Walker, L. C. (2023). Alzheimer\u2019s disease: From immunotherapy to immunoprevention. In Cell (Vol. 186, Issue 20, pp. 4260\u20134270). Elsevier BV. https:\/\/doi.org\/10.1016\/j.cell.2023.08.021
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The incurable neurodegenerative Alzheimer\u2019s disease has long been associated with \u03b2-amyloid build-up in the brain. 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